Avastin
The Avastin ejection last Monday to help overcome the effects of macular degeneration in the left eye has caused more improvement in its vision than the previous injection have made in the last 11 months. The ophthalmologist said that he would continue with the injections are long they showing improvements. My belief is that in the future I will be able to read with the left eye. We should be thankful for the progress that is being made in drugs and medical techniques.
A substance in the body called vascular endothelial growth (VEGF) factor is responsible for the growth of new blood vessels. It provokes this growth by stimulating the endothelial cells, which form the walls of the vessels and transport nutrients and oxygen to the tissues. Evidence shows that retinal pigment epithelial cells begin to wither from the lack of nutrition. The VEGF goes into action to create new vessels. This process is called “neovascularization” and it acts as a restorative function in other parts of the body. In the retina, however the vessels do not form properly, and leakage results. This leakage causes scarring in the macula eventually, loss of central vision.
Antiangiogenic drugs prevent the VEGF of a binding with the receptors on the surface of the endothelial cells. In most cases, the drug is injected into the vitreous of the eyeball, then pass into the subretinal space, where the vessels proliferate. The neovascularization is then blocked, preventing bleeding into the retina.
A substance in the body called vascular endothelial growth (VEGF) factor is responsible for the growth of new blood vessels. It provokes this growth by stimulating the endothelial cells, which form the walls of the vessels and transport nutrients and oxygen to the tissues. Evidence shows that retinal pigment epithelial cells begin to wither from the lack of nutrition. The VEGF goes into action to create new vessels. This process is called “neovascularization” and it acts as a restorative function in other parts of the body. In the retina, however the vessels do not form properly, and leakage results. This leakage causes scarring in the macula eventually, loss of central vision.
Antiangiogenic drugs prevent the VEGF of a binding with the receptors on the surface of the endothelial cells. In most cases, the drug is injected into the vitreous of the eyeball, then pass into the subretinal space, where the vessels proliferate. The neovascularization is then blocked, preventing bleeding into the retina.
posted by Russell at 7:37 AM
0 Comments:
Post a Comment
<< Home